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ijp.org

Datenschutzerklärung Verantwortlich im Sinne der Datenschutzgesetze, insbesondere der EU-Datenschutzgrundverordnung (DSGVO)Internationale Journalisten-Programme e. V. (IJP)Postfach 1565D-61455 Königstein/Taunus

indianpediatrics.net

indian pediatrics is the topmost ranking specialty journal of south east asia, the region which is currently the focus of attention for most child health programs in the world in view of its tremendous scope and potential in improving child survival. the journal emphasizes on adopting evidence based and unbiased approach towards common childhood problems.

ijpediatrics.com

international journal of contemporary pediatrics (ijcp) is an open access, international, peer-reviewed journal that publishes original research work in all areas of pediatric research. the journal's full text is available online at http://www.ijpediatrics.com. the journal allows free access to its contents. international journal of contemporary pediatrics is dedicated to publishing research in all aspects of health of infants, children, and adolescents. the journal has a broad coverage of relevant topics in pediatrics: general pediatrics, neonatal-perinatal medicine, adolescent medicine, infectious diseases, vaccines, allergy and immunology, gastroenterology, cardiology, critical care medicine, developmental-behavioral medicine, endocrinology, hematology-oncology, nephrology, neurology, emergency medicine, pulmonology, rheumatology and genetics. international journal of contemporary pediatrics (ijcp) is one of the fastest communication journals and articles are published online within short time after acceptance of manuscripts. the types of articles accepted include original research articles, review articles, insightful editorials, case reports, short communications, correspondence, images in pediatrics, clinical problem solving, perspectives and pediatric medicine. it is published monthly and available in print and online version. international journal of contemporary pediatrics (ijcp) complies with the uniform requirements for manuscripts submitted to biomedical journals, issued by the international committee for medical journal editors.

iapindia.org

indian academy of pediatrics - iap india

newbornwhocc.org

experience the joy of bringing a new life into your world with utmost care & excellent medical facilities only at the neonatal division, aiims, new delhi. our main aim through who collaborating center towards promotion of newborn care at the national and the international level.

springermedicine.com

Springer Medicine provides trustworthy, tailored, cutting-edge content for medical professionals. Follow us to stay on top of the latest advances in your field.

imrpress.com

Background: Sepsis-associated encephalopathy (SAE) is a common and severe neurological syndrome induced by sepsis and characterized by brain dysfunction. The incidence of SAE is as high as 76%. After onset, the mortality rate is as high as 60%. Even if patients survive, about 20% have long-term cognitive impairment. At present, the exact pathogenesis of SAE remains unknown. Our results showed that necroptotic protein was obviously elevated in the hippocampus of SAE mice, but the internal mechanism was still unclear. The stimulator of interferon gene (STING) protein has been reported to be a key molecule in the regulation of programmed cell death. However, the critical role of STING in the progression of SAE remains elusive. We explored the mechanism by which STING regulates hippocampal neuronal necroptosis in SAE. Methods: A cannula was embedded into the lateral ventricle of mice, and mixed lineage kinase domain-like protein (MLKL) inhibitor necrosulfonamide (NSA) and STING inhibitor C-178 were injected. The motor ability and memory function of mice after cecal ligation and puncture (CLP) were assessed by the open field test (OFT) and barnes maze test (BMT), respectively. The expression and distribution of S345p-MLKL and S366p-STING proteins in the hippocampal region of SAE mice were analyzed, and the expression levels of S366p-STING/STING, S232p-receptor-interacting protein kinase 3 (RIPK3)/RIPK3, S345p-MLKL/MLKL, and S166p-RIPK1/RIPK1 proteins in the hippocampal tissue of SAE mice were examined. Co-immunoprecipitation (CO-IP) was used to detect the relationship between STING and RIPK3 protein. Results: The surviving mice after CLP had cognitive dysfunction. The hippocampal tissues of SAE mice showed upregulated expression of S232p-RIPK3/RIPK3, S345p-MLKL/MLKL, and S166p-RIPK1/RIPK1 proteins. Mice in